CCK: The Body's Primary Short-Term Fullness Signal From the Gut

CCK is the best-characterized gastrointestinal satiety signal, released by intestinal fat and protein to reduce meal size through vagal afferents and brain CCK-A receptors — the gold standard for understanding gut-derived satiety.

Moran, Timothy H et al.·American journal of physiology. Gastrointestinal and liver physiology·2004·Moderate EvidenceReview
RPEP-00950ReviewModerate Evidence2004RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Review
Evidence
Moderate Evidence
Sample
Not reported

What This Study Found

CCK released by intestinal fat/protein is the primary short-term satiety signal, acting through vagal afferents and brain CCK-A receptors to reduce meal size — the best-characterized gut satiety peptide with therapeutic implications for obesity.

Key Numbers

How They Did This

review study on neuropeptides, gut-healing.

Why This Research Matters

Relevant for neuropeptides, gut-healing, weight-loss, receptor-signaling.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding CCK released by intestinal fat/protein is the primary short-term satiety signal, acting through vagal afferents and brain CCK-A receptors to reduce me
Evidence Grade:
moderate evidence.
Study Age:
Published in 2004.
Original Title:
Gastrointestinal satiety signals II. Cholecystokinin.
Published In:
American journal of physiology. Gastrointestinal and liver physiology, 286(2), G183-8 (2004)
Database ID:
RPEP-00950

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study

Summarizes existing research on a topic.

What do these levels mean? →

Frequently Asked Questions

What was studied?

CCK: The Body's Primary Short-Term Fullness Signal From the Gut

What was found?

CCK is the best-characterized gastrointestinal satiety signal, released by intestinal fat and protein to reduce meal size through vagal afferents and brain CCK-A receptors — the gold standard for understanding gut-derived satiety.

Read More on RethinkPeptides

Cite This Study

RPEP-00950·https://rethinkpeptides.com/research/RPEP-00950

APA

Moran, Timothy H; Kinzig, Kimberly P. (2004). Gastrointestinal satiety signals II. Cholecystokinin.. American journal of physiology. Gastrointestinal and liver physiology, 286(2), G183-8.

MLA

Moran, Timothy H, et al. "Gastrointestinal satiety signals II. Cholecystokinin.." American journal of physiology. Gastrointestinal and liver physiology, 2004.

RethinkPeptides

RethinkPeptides Research Database. "Gastrointestinal satiety signals II. Cholecystokinin." RPEP-00950. Retrieved from https://rethinkpeptides.com/research/moran-2004-gastrointestinal-satiety-signals-ii

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.