Digested Proteins Release Peptides That Trigger CCK From Gut Hormone Cells

Protein hydrolysates directly stimulated CCK release from enteroendocrine cells and acted as partial CCK1 receptor agonists, revealing how protein digestion products directly trigger satiety hormone secretion.

Foltz, Martin et al.·Journal of agricultural and food chemistry·2008·Preliminary Evidencein-vitro
RPEP-01338In VitroPreliminary Evidence2008RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
in-vitro
Evidence
Preliminary Evidence
Sample
Not reported

What This Study Found

Protein hydrolysates directly stimulated CCK secretion from STC-1 enteroendocrine cells and showed partial CCK1 receptor agonist activity, demonstrating dual mechanisms: dietary peptides both trigger satiety hormone release AND directly activate satiety receptors.

Key Numbers

How They Did This

in-vitro study.

Why This Research Matters

Relevant for bioactive-food-peptides, neuropeptides, weight-loss.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding Protein hydrolysates directly stimulated CCK secretion from STC-1 enteroendocrine cells and showed partial CCK1 receptor agonist activity, demonstrati
Evidence Grade:
preliminary evidence.
Study Age:
Published in 2008.
Original Title:
Protein hydrolysates induce CCK release from enteroendocrine cells and act as partial agonists of the CCK1 receptor.
Published In:
Journal of agricultural and food chemistry, 56(3), 837-43 (2008)
Database ID:
RPEP-01338

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
What do these levels mean? →

Frequently Asked Questions

What was studied?

Digested Proteins Release Peptides That Trigger CCK From Gut Hormone Cells

What was found?

Protein hydrolysates directly stimulated CCK release from enteroendocrine cells and acted as partial CCK1 receptor agonists, revealing how protein digestion products directly trigger satiety hormone secretion.

Read More on RethinkPeptides

Cite This Study

RPEP-01338·https://rethinkpeptides.com/research/RPEP-01338

APA

Foltz, Martin; Ansems, Patrick; Schwarz, Jessica; Tasker, Maria C; Lourbakos, Afrodite; Gerhardt, Cindy C. (2008). Protein hydrolysates induce CCK release from enteroendocrine cells and act as partial agonists of the CCK1 receptor.. Journal of agricultural and food chemistry, 56(3), 837-43. https://doi.org/10.1021/jf072611h

MLA

Foltz, Martin, et al. "Protein hydrolysates induce CCK release from enteroendocrine cells and act as partial agonists of the CCK1 receptor.." Journal of agricultural and food chemistry, 2008. https://doi.org/10.1021/jf072611h

RethinkPeptides

RethinkPeptides Research Database. "Protein hydrolysates induce CCK release from enteroendocrine..." RPEP-01338. Retrieved from https://rethinkpeptides.com/research/foltz-2008-protein-hydrolysates-induce-cck

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.