rethinkPeptides Search
Menu
Study breakdown

VIP Expands Its Immune Role: Controlling Th17 Cells in Addition to Th1 and Tregs

ReviewModerate evidence
The takeaway

VIP regulation of immune responses expanded beyond Th1/Treg balance to include Th17 cell modulation, with VIP suppressing pathogenic Th17 differentiation — broadening its anti-autoimmune therapeutic potential.

Key finding

VIP's immunomodulatory spectrum expanded to include Th17 cell regulation: suppressing pathogenic IL-17 producing cells in addition to its established

What the researchers found

VIP's immunomodulatory spectrum expanded to include Th17 cell regulation: suppressing pathogenic IL-17 producing cells in addition to its established Th1 suppression and Treg promotion — comprehensive anti-autoimmune peptide with Th1/Th17/Treg triple regulation.

Why it matters

Relevant for neuropeptides, immune-function, inflammation.

How the study worked

review study.

What this study cannot tell us

See abstract.

How to read the evidence

moderate evidence.

When this study was published

Published in 2008.

The bigger picture

Advances peptide research.

Questions still open

  • Further research needed.
  • Clinical translation to evaluate.

Common questions

What was studied?
VIP Expands Its Immune Role: Controlling Th17 Cells in Addition to Th1 and Tregs
What was found?
VIP regulation of immune responses expanded beyond Th1/Treg balance to include Th17 cell modulation, with VIP suppressing pathogenic Th17 differentiation — broadening its anti-autoimmune therapeutic potential.

Read the original research

Vasoactive intestinal peptide-mediated Th17 differentiation: an expanding spectrum of vasoactive intestinal peptide effects in immunity and autoimmunity.

Annals of the New York Academy of Sciences, 1144, 83-9

Citation

Yadav, Mahesh; Goetzl, Edward J. (2008). Vasoactive intestinal peptide-mediated Th17 differentiation: an expanding spectrum of vasoactive intestinal peptide effects in immunity and autoimmunity.. Annals of the New York Academy of Sciences, 1144, 83-9. https://doi.org/10.1196/annals.1418.020