VIP Peptide Suppresses the Dangerous Th17 Immune Response in Autoimmune Disease

VIP (vasoactive intestinal peptide) specifically regulated Th17 cell function in autoimmune inflammation, suppressing the IL-17-producing cells that drive multiple autoimmune diseases — a targeted anti-autoimmune mechanism.

Leceta, Javier et al.·Neuroimmunomodulation·2007·Moderate EvidenceAnimal StudyAnimal Study
RPEP-01256Animal StudyModerate Evidence2007RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Animal Study
Evidence
Moderate Evidence
Sample
Not reported

What This Study Found

VIP suppressed Th17 cell differentiation and IL-17 production in autoimmune inflammation models, specifically targeting the pathogenic Th17 response that drives rheumatoid arthritis, EAE (MS model), and other autoimmune diseases — precision anti-autoimmune peptide therapy.

Key Numbers

How They Did This

animal-study study on neuropeptides, inflammation.

Why This Research Matters

Relevant for neuropeptides, inflammation, immune-function.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding VIP suppressed Th17 cell differentiation and IL-17 production in autoimmune inflammation models, specifically targeting the pathogenic Th17 response t
Evidence Grade:
moderate evidence.
Study Age:
Published in 2007.
Original Title:
Vasoactive intestinal peptide regulates Th17 function in autoimmune inflammation.
Published In:
Neuroimmunomodulation, 14(3-4), 134-8 (2007)
Database ID:
RPEP-01256

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / Observational
Case Report / Animal StudyOne case or non-human subjects
This study

Tests effects in animals (usually mice or rats), not humans.

What do these levels mean? →

Frequently Asked Questions

What was studied?

VIP Peptide Suppresses the Dangerous Th17 Immune Response in Autoimmune Disease

What was found?

VIP (vasoactive intestinal peptide) specifically regulated Th17 cell function in autoimmune inflammation, suppressing the IL-17-producing cells that drive multiple autoimmune diseases — a targeted anti-autoimmune mechanism.

Read More on RethinkPeptides

Cite This Study

RPEP-01256·https://rethinkpeptides.com/research/RPEP-01256

APA

Leceta, Javier; Gomariz, Rosa P; Martinez, Carmen; Carrión, Mar; Arranz, Alicia; Juarranz, Yasmina. (2007). Vasoactive intestinal peptide regulates Th17 function in autoimmune inflammation.. Neuroimmunomodulation, 14(3-4), 134-8.

MLA

Leceta, Javier, et al. "Vasoactive intestinal peptide regulates Th17 function in autoimmune inflammation.." Neuroimmunomodulation, 2007.

RethinkPeptides

RethinkPeptides Research Database. "Vasoactive intestinal peptide regulates Th17 function in aut..." RPEP-01256. Retrieved from https://rethinkpeptides.com/research/leceta-2007-vasoactive-intestinal-peptide-regulates

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.