Oral gelatin hydrolysate increased postprandial GLP-1, insulin, and glucagon while suppressing ghrelin in both normal and obese subjects — collagen peptides trigger satiety hormones beyond their protein content.
Key findingOral gelatin hydrolysate meals stimulated GLP-1, insulin, and glucagon release while suppressing ghrelin in both normal weight and obese subjects, dem
What the researchers found
Oral gelatin hydrolysate meals stimulated GLP-1, insulin, and glucagon release while suppressing ghrelin in both normal weight and obese subjects, demonstrating collagen peptides trigger satiety hormone responses beyond simple protein-mediated effects.
Why it matters
Relevant for collagen-peptides, neuropeptides, weight-loss.
How the study worked
clinical-trial study.
What this study cannot tell us
See abstract.
How to read the evidence
moderate evidence.
When this study was published
Published in 2008.
The bigger picture
Advances peptide research.
Questions still open
- Further research needed.
- Clinical translation to evaluate.
Common questions
What was studied?
What was found?
Read the original research
Oral ingestion of a hydrolyzed gelatin meal in subjects with normal weight and in obese patients: Postprandial effect on circulating gut peptides, glucose and insulin.
Eating and weight disorders : EWD, 13(1), 48-53
Citation
Rubio, I G; Castro, G; Zanini, A C; Medeiros-Neto, G. (2008). Oral ingestion of a hydrolyzed gelatin meal in subjects with normal weight and in obese patients: Postprandial effect on circulating gut peptides, glucose and insulin.. Eating and weight disorders : EWD, 13(1), 48-53.