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Study breakdown

Lactoferricin Kills Listeria-Infected Human Immune Cells by Triggering Their Suicide Program

In VitroPreliminary evidence
The takeaway

Bovine lactoferricin induced apoptosis of Listeria-infected human macrophages while sparing uninfected cells — selectively eliminating the infected cell reservoir to control intracellular infection.

Key finding

Lactoferricin B selectively induced apoptosis of Listeria monocytogenes-infected macrophages while sparing uninfected cells, eliminating the intracell

What the researchers found

Lactoferricin B selectively induced apoptosis of Listeria monocytogenes-infected macrophages while sparing uninfected cells, eliminating the intracellular bacterial reservoir — a selective anti-infection strategy targeting infected immune cells.

Why it matters

Relevant for antimicrobial-peptides, infection, immune-function.

How the study worked

in-vitro study on antimicrobial-peptides, infection.

What this study cannot tell us

See abstract.

How to read the evidence

preliminary evidence.

When this study was published

Published in 2005.

The bigger picture

Advances peptide research with clinical implications.

Questions still open

  • Further research needed.
  • Clinical translation to evaluate.

Common questions

What was studied?
Lactoferricin Kills Listeria-Infected Human Immune Cells by Triggering Their Suicide Program
What was found?
Bovine lactoferricin induced apoptosis of Listeria-infected human macrophages while sparing uninfected cells — selectively eliminating the infected cell reservoir to control intracellular infection.

Read the original research

Apoptotic death of Listeria monocytogenes-infected human macrophages induced by lactoferricin B, a bovine lactoferrin-derived peptide.

International journal of immunopathology and pharmacology, 18(2), 317-25

Citation

Longhi, C; Conte, M P; Ranaldi, S; Penta, M; Valenti, P; Tinari, A; Superti, F; Seganti, L. (2005). Apoptotic death of Listeria monocytogenes-infected human macrophages induced by lactoferricin B, a bovine lactoferrin-derived peptide.. International journal of immunopathology and pharmacology, 18(2), 317-25.