Semax Specifically Binds Brain Cells and Increases BDNF Protein Levels

Semax showed specific binding to rat brain cells and increased BDNF protein levels, confirming it has a dedicated brain receptor and works through neurotrophin enhancement for cognitive and neuroprotective effects.

Dolotov, Oleg V et al.·Journal of neurochemistry·2006·Moderate EvidenceAnimal StudyAnimal Study
RPEP-01130Animal StudyModerate Evidence2006RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Animal Study
Evidence
Moderate Evidence
Sample
Not reported

What This Study Found

Semax (ACTH 4-10 analog) showed specific binding to rat brain tissue and increased brain-derived neurotrophic factor (BDNF) protein levels, confirming receptor-mediated neurotrophin enhancement as the mechanism for its nootropic and neuroprotective activities.

Key Numbers

How They Did This

animal-study study on semax, neuroprotection.

Why This Research Matters

Relevant for semax, neuroprotection, cognitive-enhancement.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding Semax (ACTH 4-10 analog) showed specific binding to rat brain tissue and increased brain-derived neurotrophic factor (BDNF) protein levels, confirming
Evidence Grade:
moderate evidence.
Study Age:
Published in 2006.
Original Title:
Semax, an analogue of adrenocorticotropin (4-10), binds specifically and increases levels of brain-derived neurotrophic factor protein in rat basal forebrain.
Published In:
Journal of neurochemistry, 97 Suppl 1, 82-6 (2006)
Database ID:
RPEP-01130

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / Observational
Case Report / Animal StudyOne case or non-human subjects
This study

Tests effects in animals (usually mice or rats), not humans.

What do these levels mean? →

Frequently Asked Questions

What was studied?

Semax Specifically Binds Brain Cells and Increases BDNF Protein Levels

What was found?

Semax showed specific binding to rat brain cells and increased BDNF protein levels, confirming it has a dedicated brain receptor and works through neurotrophin enhancement for cognitive and neuroprotective effects.

Read More on RethinkPeptides

Cite This Study

RPEP-01130·https://rethinkpeptides.com/research/RPEP-01130

APA

Dolotov, Oleg V; Karpenko, Ekaterina A; Seredenina, Tamara S; Inozemtseva, Lyudmila S; Levitskaya, Natalia G; Zolotarev, Yuriy A; Kamensky, Andrey A; Grivennikov, Igor A; Engele, Juergen; Myasoedov, Nikolay F. (2006). Semax, an analogue of adrenocorticotropin (4-10), binds specifically and increases levels of brain-derived neurotrophic factor protein in rat basal forebrain.. Journal of neurochemistry, 97 Suppl 1, 82-6.

MLA

Dolotov, Oleg V, et al. "Semax, an analogue of adrenocorticotropin (4-10), binds specifically and increases levels of brain-derived neurotrophic factor protein in rat basal forebrain.." Journal of neurochemistry, 2006.

RethinkPeptides

RethinkPeptides Research Database. "Semax, an analogue of adrenocorticotropin (4-10), binds spec..." RPEP-01130. Retrieved from https://rethinkpeptides.com/research/dolotov-2006-semax-an-analogue-of

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.