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Study breakdown

Who Benefits Most from GLP-1 Drugs for Quitting Smoking? First Clues from a Pilot Trial

Secondary Analysis Of Randomized Controlled TrialModerate evidence
The takeaway

A pilot trial found the GLP-1 drug exenatide worked best for smoking cessation in heavy smokers without obesity or depression who carry a specific nicotine receptor gene variant.

94.4% probability

Bayesian analysis showed 94.4% probability that exenatide is more effective than placebo for smoking cessation in participants without obesity

What the researchers found

In a secondary analysis of a pilot RCT, the GLP-1 agonist exenatide showed stronger smoking cessation benefits in specific patient subgroups. Exenatide (added to nicotine patch) worked better than placebo plus nicotine patch in people who smoked more than 20 cigarettes per day (posterior probability 81.7%), those without obesity (PP = 94.4%), those without prediabetes (PP = 76.0%), and those with no or minimal depression (PP = 91.2%).

A striking genetic finding emerged: exenatide was more effective only in individuals with the CHRNA rs16969968 GG genotype (PP = 88.6%), a nicotine receptor gene variant associated with smoking behavior. This suggests that GLP-1 drugs for smoking cessation may be most effective in a specific subset of smokers — heavy smokers who are not obese, not depressed, and carry a particular genetic profile.

Why it matters

The idea that GLP-1 drugs could help people quit smoking is one of the most exciting emerging applications of these peptides. But not everyone may benefit equally. This study is among the first to identify who responds best to GLP-1 treatment for smoking cessation, potentially enabling precision medicine approaches. The finding that metabolic health, depression status, and genetics all predict response suggests the mechanism involves more than just appetite suppression.

The numbers in context

n=84 · Exenatide 2 mg SC once weekly + nicotine patch 21 mg · Stronger in >20 cig/day (PP=81.7%) · Stronger without obesity (PP=94.4%) · Only effective with minimal depression (PP=91.2%) · CHRNA rs16969968 GG genotype (PP=88.6%)

How the study worked

Secondary analysis of a randomized, placebo-controlled pilot trial. 84 smokers with prediabetes and/or overweight were randomized 1:1 to once-weekly exenatide 2 mg or placebo, both with nicotine patch (21 mg) and counseling. The primary outcome was biologically confirmed 7-day point prevalence abstinence at week 6. Bayesian generalized linear modeling was used to explore how baseline demographics, clinical factors, smoking intensity, psychosocial measures, and genetic variants moderated treatment response.

Who was studied

Adult smokers with prediabetes and/or overweight/obesity

What this study cannot tell us

This is a secondary (post-hoc) analysis of a small pilot trial with only 84 participants — subgroup analyses with this sample size should be considered hypothesis-generating, not definitive. Bayesian posterior probabilities ≥75% were used as thresholds, which is less stringent than conventional significance testing. Multiple subgroup comparisons increase the risk of finding spurious associations. The parent trial used exenatide (an older GLP-1 agonist), not semaglutide or tirzepatide.

How to read the evidence

This is a well-designed secondary analysis of a randomized controlled trial published in a leading tobacco research journal. However, the small sample size (n=84) and post-hoc nature of the subgroup analyses mean findings should be considered preliminary and hypothesis-generating.

When this study was published

Published in 2025. Highly current research at the frontier of GLP-1 applications for addictive behavior, with the parent trial being one of the first RCTs of GLP-1 agonists for smoking cessation.

The bigger picture

GLP-1 agonists are increasingly linked to reduced addictive behaviors — from alcohol to smoking to compulsive eating. This study moves the field toward precision by identifying which smokers are most likely to benefit. If confirmed in larger trials, genetic testing and clinical profiling could guide who gets prescribed GLP-1 drugs for smoking cessation, mirroring the precision medicine revolution in cancer treatment.

Questions still open

  • Would newer, more potent GLP-1 agonists like semaglutide show even stronger smoking cessation effects in these responder subgroups?
  • Why does exenatide work better in non-obese smokers — does the mechanism differ from its weight-loss effects?
  • Could the CHRNA rs16969968 genotype become a clinical biomarker for prescribing GLP-1 drugs for smoking cessation?

Common questions

Can GLP-1 drugs like Ozempic help you quit smoking?
Early evidence is promising. This pilot trial found that exenatide (an older GLP-1 drug) added to nicotine patches improved smoking abstinence compared to patches alone. However, the benefit wasn't universal — it was strongest in heavy smokers without obesity or depression. GLP-1 drugs are not yet approved for smoking cessation, and larger trials are needed.
Why would a diabetes drug help with smoking?
GLP-1 receptors exist not just in the pancreas but throughout the brain, including reward centers. Researchers believe GLP-1 drugs may reduce the rewarding effects of nicotine, similar to how they reduce food cravings. The finding that a specific nicotine receptor gene variant predicts response suggests GLP-1 drugs interact with the same brain pathways involved in nicotine addiction.

Read the original research

Exploring Predictors of Treatment Response to GLP-1 Receptor Agonists for Smoking Cessation.

Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco, 27(7), 1294-1300

Citation

Yammine, Luba; de Dios, Constanza; Suchting, Robert; Green, Charles E; Nielsen, David A; Walss-Bass, Consuelo; Schmitz, Joy M. (2025). Exploring Predictors of Treatment Response to GLP-1 Receptor Agonists for Smoking Cessation.. Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco, 27(7), 1294-1300. https://doi.org/10.1093/ntr/ntaf005