VIP Receptor Expression on Immune Cells Is Dynamically Regulated by Inflammatory Signals

VPAC-1 receptor expression on primary mouse macrophages was regulated by both stimulatory (LPS, IFN-γ) and suppressive (IL-10, TGF-β) signals, explaining how the VIP anti-inflammatory system adapts to inflammatory context.

Vomhof-DeKrey, Emilie E et al.·Brain·2008·Preliminary Evidencein-vitro
RPEP-01434In VitroPreliminary Evidence2008RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
in-vitro
Evidence
Preliminary Evidence
Sample
Not reported

What This Study Found

VPAC-1 receptor on macrophages was upregulated by pro-inflammatory signals (LPS, IFN-γ) and downregulated by anti-inflammatory signals (IL-10, TGF-β) — dynamic receptor regulation that adapts VIP anti-inflammatory sensitivity to the inflammatory microenvironment.

Key Numbers

How They Did This

in-vitro study.

Why This Research Matters

Relevant for neuropeptides, immune-function.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding VPAC-1 receptor on macrophages was upregulated by pro-inflammatory signals (LPS, IFN-γ) and downregulated by anti-inflammatory signals (IL-10, TGF-β)
Evidence Grade:
preliminary evidence.
Study Age:
Published in 2008.
Original Title:
Stimulatory and suppressive signal transduction regulates vasoactive intestinal peptide receptor-1 (VPAC-1) in primary mouse CD4 T cells.
Published In:
Brain, behavior, and immunity, 22(7), 1024-1031 (2008)
Database ID:
RPEP-01434

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
What do these levels mean? →

Frequently Asked Questions

What was studied?

VIP Receptor Expression on Immune Cells Is Dynamically Regulated by Inflammatory Signals

What was found?

VPAC-1 receptor expression on primary mouse macrophages was regulated by both stimulatory (LPS, IFN-γ) and suppressive (IL-10, TGF-β) signals, explaining how the VIP anti-inflammatory system adapts to inflammatory context.

Read More on RethinkPeptides

Cite This Study

RPEP-01434·https://rethinkpeptides.com/research/RPEP-01434

APA

Vomhof-DeKrey, Emilie E; Dorsam, Glenn Paul. (2008). Stimulatory and suppressive signal transduction regulates vasoactive intestinal peptide receptor-1 (VPAC-1) in primary mouse CD4 T cells.. Brain, behavior, and immunity, 22(7), 1024-1031. https://doi.org/10.1016/j.bbi.2008.04.006

MLA

Vomhof-DeKrey, Emilie E, et al. "Stimulatory and suppressive signal transduction regulates vasoactive intestinal peptide receptor-1 (VPAC-1) in primary mouse CD4 T cells.." Brain, 2008. https://doi.org/10.1016/j.bbi.2008.04.006

RethinkPeptides

RethinkPeptides Research Database. "Stimulatory and suppressive signal transduction regulates va..." RPEP-01434. Retrieved from https://rethinkpeptides.com/research/vomhof-dekrey-2008-stimulatory-and-suppressive-signal

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.