Attenuation of nociceptive and paclitaxel-induced neuropathic pain by targeting inflammatory, CGRP and substance P signaling using 3-Hydroxyflavone.

Ullah, Rahim et al.·Neurochemistry international·2021·Preliminary Evidenceanimal
RPEP-05834AnimalPreliminary Evidence2021RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
animal
Evidence
Preliminary Evidence
Sample
Mice (pain tests) and Sprague Dawley rats (neuropathy model)
Participants
Mice (pain tests) and Sprague Dawley rats (neuropathy model)

What This Study Found

3-Hydroxyflavone reduced paclitaxel-induced neuropathic pain in rats by suppressing spinal cord CGRP, substance P, and inflammatory cytokines, with NK1R as the likely primary target.

Key Numbers

Paclitaxel 4 mg/kg days 1,3,5,7; reduced TNF-a, IL-1b, IL-6, CGRP, substance P mRNA; NK1R predicted strongest binding target

How They Did This

Animal study. Induced peripheral neuropathy with paclitaxel (4 mg/kg on days 1, 3, 5, 7) in rats. Treated with 3HF. Measured pain behaviors, spinal cord gene expression (RT-PCR), and computational docking.

Why This Research Matters

Chemotherapy-induced nerve pain has no good treatments. A compound that targets multiple pain pathways could be more effective than current options.

What This Study Doesn't Tell Us

Animal study in rats. Computational docking is prediction, not proven binding. 3HF has not been tested in humans for this use. Specific dose not detailed.

Trust & Context

Original Title:
Attenuation of nociceptive and paclitaxel-induced neuropathic pain by targeting inflammatory, CGRP and substance P signaling using 3-Hydroxyflavone.
Published In:
Neurochemistry international, 144, 104981 (2021)
Database ID:
RPEP-05834

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
What do these levels mean? →

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Cite This Study

RPEP-05834·https://rethinkpeptides.com/research/RPEP-05834

APA

Ullah, Rahim; Ali, Gowhar; Subhan, Fazal; Naveed, Muhammad; Khan, Ajmal; Khan, Jawad; Halim, Sobia Ahsan; Ahmad, Nisar; Zakiullah; Al-Harrasi, Ahmed. (2021). Attenuation of nociceptive and paclitaxel-induced neuropathic pain by targeting inflammatory, CGRP and substance P signaling using 3-Hydroxyflavone.. Neurochemistry international, 144, 104981. https://doi.org/10.1016/j.neuint.2021.104981

MLA

Ullah, Rahim, et al. "Attenuation of nociceptive and paclitaxel-induced neuropathic pain by targeting inflammatory, CGRP and substance P signaling using 3-Hydroxyflavone.." Neurochemistry international, 2021. https://doi.org/10.1016/j.neuint.2021.104981

RethinkPeptides

RethinkPeptides Research Database. "Attenuation of nociceptive and paclitaxel-induced neuropathi..." RPEP-05834. Retrieved from https://rethinkpeptides.com/research/ullah-2021-attenuation-of-nociceptive-and

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.