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Study breakdown

AAV gene therapy delivering NPY and Y2 receptor to hippocampus reduces seizures in rodents and reaches baboon brain

evidence
The takeaway

AAV-mediated delivery of neuropeptide Y (NPY) and Y2 receptor to hippocampus reduced seizures in both rapid kindling and genetic epilepsy rodent models, with successful MRI-guided delivery to baboon hippocampus and no adverse events.

Baboon brain successfully reached

NPY gene therapy reduced seizures in 2 rodent models AND achieved MRI-guided delivery to baboon hippocampus with no adverse events—a critical step toward human clinical trials

What the researchers found

SPK100.NPY-Y2R: ↓neuronal activity in cultures + slices. ↓Seizure progression/duration (rat kindling). ↓Spontaneous seizures (genetic mouse model). MRI-guided delivery → successful baboon hippocampus transduction. No adverse events. Non-human primate step completed.

Why it matters

30% of epilepsy patients resist all current drugs. NPY gene therapy could provide lasting seizure control from a single treatment, and successful baboon delivery brings it closer to human trials.

How the study worked

In vitro: rat cortical cultures, mouse hippocampal slices. In vivo: rat rapid kindling, synapsin triple KO mice spontaneous seizures. Non-human primate: MRI-guided CED to baboon hippocampus.

What this study cannot tell us

Rodent models. Baboon delivery confirmed but no seizure testing in primates. Long-term NPY expression durability unknown. Neurosurgical delivery carries inherent risks.

How to read the evidence

Comprehensive preclinical program spanning in vitro, multiple rodent models, and non-human primate delivery. Strong translational evidence.

When this study was published

Published in 2025.

The bigger picture

NPY gene therapy represents a potential cure for drug-resistant temporal lobe epilepsy—the most common type. A single neurosurgical procedure delivering lasting NPY expression could replace lifelong medication or invasive surgery.

Questions still open

  • When will human clinical trials of AAV.NPY-Y2R begin?
  • How long does NPY expression persist from a single AAV delivery?
  • Would ectopic NPY expression cause appetite or other side effects?

Common questions

Could gene therapy cure epilepsy?
This research brings us closer. By delivering the gene for neuropeptide Y (a natural seizure-suppressing peptide) directly to the hippocampus, seizures were reduced in two different epilepsy models. The successful delivery to a baboon brain with no side effects is a critical step toward human trials.
How is NPY gene therapy different from anti-seizure drugs?
Current drugs must be taken daily and do not work for 30% of patients. NPY gene therapy could provide lasting seizure control from a single neurosurgical procedure by making the brain produce its own seizure-suppressing peptide permanently. It directly treats the brain region where seizures originate.

Read the original research

AAV-mediated gene therapy for focal epilepsy by expressing neuropeptide Y and Y2 receptor in rodent and non-human primate hippocampus.

Molecular therapy : the journal of the American Society of Gene Therapy, 33(9), 4239-4258

Citation

Terzic, Barbara; Melin, Esbjörn; Fagergren, Pernilla; Dobry, David; Cattaneo, Stefano; Giupponi, Iris; Bettegazzi, Barbara; Simonato, Michele; Agerman, Karin; Kokaia, Merab; Moon, Lawrence; Ramsburg, Elizabeth. (2025). AAV-mediated gene therapy for focal epilepsy by expressing neuropeptide Y and Y2 receptor in rodent and non-human primate hippocampus.. Molecular therapy : the journal of the American Society of Gene Therapy, 33(9), 4239-4258. https://doi.org/10.1016/j.ymthe.2025.06.019