An Arachidonic Acid Metabolite Kills Pain by Releasing Beta-Endorphin and Met-Enkephalin

The fatty acid metabolite 14,15-EET produced pain relief by triggering release of beta-endorphin and met-enkephalin, establishing a lipid-opioid peptide connection for pain modulation.

Terashvili, Maia et al.·The Journal of pharmacology and experimental therapeutics·2008·Preliminary EvidenceAnimal StudyAnimal Study
RPEP-01429Animal StudyPreliminary Evidence2008RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Animal Study
Evidence
Preliminary Evidence
Sample
Not reported

What This Study Found

14,15-Epoxyeicosatrienoic acid (EET) produced antinociception mediated by beta-endorphin and met-enkephalin release (confirmed by antisera blocking), establishing a lipid mediator → endogenous opioid peptide → pain relief signaling cascade.

Key Numbers

How They Did This

animal-study study.

Why This Research Matters

Relevant for opioid-peptides, pain, neuropeptides.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding 14,15-Epoxyeicosatrienoic acid (EET) produced antinociception mediated by beta-endorphin and met-enkephalin release (confirmed by antisera blocking),
Evidence Grade:
preliminary evidence.
Study Age:
Published in 2008.
Original Title:
Antinociception produced by 14,15-epoxyeicosatrienoic acid is mediated by the activation of beta-endorphin and met-enkephalin in the rat ventrolateral periaqueductal gray.
Published In:
The Journal of pharmacology and experimental therapeutics, 326(2), 614-22 (2008)
Database ID:
RPEP-01429

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / Observational
Case Report / Animal StudyOne case or non-human subjects
This study

Tests effects in animals (usually mice or rats), not humans.

What do these levels mean? →

Frequently Asked Questions

What was studied?

An Arachidonic Acid Metabolite Kills Pain by Releasing Beta-Endorphin and Met-Enkephalin

What was found?

The fatty acid metabolite 14,15-EET produced pain relief by triggering release of beta-endorphin and met-enkephalin, establishing a lipid-opioid peptide connection for pain modulation.

Read More on RethinkPeptides

Cite This Study

RPEP-01429·https://rethinkpeptides.com/research/RPEP-01429

APA

Terashvili, Maia; Tseng, Leon F; Wu, Hsiang-En; Narayanan, Jayashree; Hart, Lucas M; Falck, John R; Pratt, Phillip F; Harder, David R. (2008). Antinociception produced by 14,15-epoxyeicosatrienoic acid is mediated by the activation of beta-endorphin and met-enkephalin in the rat ventrolateral periaqueductal gray.. The Journal of pharmacology and experimental therapeutics, 326(2), 614-22. https://doi.org/10.1124/jpet.108.136739

MLA

Terashvili, Maia, et al. "Antinociception produced by 14,15-epoxyeicosatrienoic acid is mediated by the activation of beta-endorphin and met-enkephalin in the rat ventrolateral periaqueductal gray.." The Journal of pharmacology and experimental therapeutics, 2008. https://doi.org/10.1124/jpet.108.136739

RethinkPeptides

RethinkPeptides Research Database. "Antinociception produced by 14,15-epoxyeicosatrienoic acid i..." RPEP-01429. Retrieved from https://rethinkpeptides.com/research/terashvili-2008-antinociception-produced-by-1415epoxyeicosatrienoic

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.