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Study breakdown

Oxidative Stress Reduces Adiponectin in Heart Failure: Another Piece of the Metabolic Puzzle

Cross SectionalModerate evidence
The takeaway

Oxidative stress markers correlated inversely with adiponectin in heart failure patients, with reduced adiponectin associated with worse outcomes — oxidative stress suppresses this protective adipokine.

Key finding

Plasma oxidative stress markers inversely correlated with adiponectin levels in CHF patients, with low adiponectin associated with worse outcomes — ox

What the researchers found

Plasma oxidative stress markers inversely correlated with adiponectin levels in CHF patients, with low adiponectin associated with worse outcomes — oxidative stress suppresses the protective adipokine adiponectin, adding metabolic dysfunction to heart failure pathophysiology.

Why it matters

Relevant for cardiovascular.

How the study worked

cross-sectional study.

What this study cannot tell us

See abstract.

How to read the evidence

moderate evidence.

When this study was published

Published in 2008.

The bigger picture

Advances peptide research.

Questions still open

  • Further research needed.
  • Clinical translation to evaluate.

Common questions

What was studied?
Oxidative Stress Reduces Adiponectin in Heart Failure: Another Piece of the Metabolic Puzzle
What was found?
Oxidative stress markers correlated inversely with adiponectin in heart failure patients, with reduced adiponectin associated with worse outcomes — oxidative stress suppresses this protective adipokine.

Read the original research

Impact of oxidative stress on plasma adiponectin in patients with chronic heart failure.

Circulation journal : official journal of the Japanese Circulation Society, 72(4), 563-8

Citation

Tanaka, Toshinari; Tsutamoto, Takayoshi; Nishiyama, Keizo; Sakai, Hiroshi; Fujii, Masanori; Yamamoto, Takashi; Horie, Minoru. (2008). Impact of oxidative stress on plasma adiponectin in patients with chronic heart failure.. Circulation journal : official journal of the Japanese Circulation Society, 72(4), 563-8.