Prolactin-Releasing Peptide Naturally Reduces Food Intake in Rodents

Endogenous prolactin-releasing peptide (PrRP) regulated food intake in rodents, with PrRP-deficient mice showing increased body weight and food intake — establishing PrRP as a natural satiety signal beyond its prolactin-releasing function.

Takayanagi, Yuki et al.·The Journal of clinical investigation·2008·Moderate EvidenceAnimal StudyAnimal Study
RPEP-01427Animal StudyModerate Evidence2008RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Animal Study
Evidence
Moderate Evidence
Sample
Not reported

What This Study Found

PrRP-deficient mice showed increased food intake and body weight, while PrRP administration reduced feeding, establishing endogenous prolactin-releasing peptide as a novel satiety signal acting through hypothalamic and brainstem appetite circuits.

Key Numbers

How They Did This

animal-study study.

Why This Research Matters

Relevant for neuropeptides, weight-loss.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding PrRP-deficient mice showed increased food intake and body weight, while PrRP administration reduced feeding, establishing endogenous prolactin-releasi
Evidence Grade:
moderate evidence.
Study Age:
Published in 2008.
Original Title:
Endogenous prolactin-releasing peptide regulates food intake in rodents.
Published In:
The Journal of clinical investigation, 118(12), 4014-24 (2008)
Database ID:
RPEP-01427

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / Observational
Case Report / Animal StudyOne case or non-human subjects
This study

Tests effects in animals (usually mice or rats), not humans.

What do these levels mean? →

Frequently Asked Questions

What was studied?

Prolactin-Releasing Peptide Naturally Reduces Food Intake in Rodents

What was found?

Endogenous prolactin-releasing peptide (PrRP) regulated food intake in rodents, with PrRP-deficient mice showing increased body weight and food intake — establishing PrRP as a natural satiety signal beyond its prolactin-releasing function.

Read More on RethinkPeptides

Cite This Study

RPEP-01427·https://rethinkpeptides.com/research/RPEP-01427

APA

Takayanagi, Yuki; Matsumoto, Hirokazu; Nakata, Masanori; Mera, Takashi; Fukusumi, Shoji; Hinuma, Shuji; Ueta, Yoichi; Yada, Toshihiko; Leng, Gareth; Onaka, Tatsushi. (2008). Endogenous prolactin-releasing peptide regulates food intake in rodents.. The Journal of clinical investigation, 118(12), 4014-24. https://doi.org/10.1172/JCI34682

MLA

Takayanagi, Yuki, et al. "Endogenous prolactin-releasing peptide regulates food intake in rodents.." The Journal of clinical investigation, 2008. https://doi.org/10.1172/JCI34682

RethinkPeptides

RethinkPeptides Research Database. "Endogenous prolactin-releasing peptide regulates food intake..." RPEP-01427. Retrieved from https://rethinkpeptides.com/research/takayanagi-2008-endogenous-prolactinreleasing-peptide-regulates

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.