Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury.

RPEP-018692011RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Not classified
Evidence
Not graded
Sample
Not reported

What This Study Found

Key Numbers

How They Did This

Why This Research Matters

What This Study Doesn't Tell Us

Trust & Context

Original Title:
Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury.
Published In:
Journal of the American Society of Nephrology : JASN, 22(6), 1041-52 (2011)
Database ID:
RPEP-01869

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
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Cite This Study

RPEP-01869·https://rethinkpeptides.com/research/RPEP-01869

APA

Szeto, Hazel H; Liu, Shaoyi; Soong, Yi; Wu, Dunli; Darrah, Shaun F; Cheng, Feng-Ying; Zhao, Zhihong; Ganger, Michael; Tow, Clara Y; Seshan, Surya V. (2011). Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury.. Journal of the American Society of Nephrology : JASN, 22(6), 1041-52. https://doi.org/10.1681/ASN.2010080808

MLA

Szeto, Hazel H, et al. "Mitochondria-targeted peptide accelerates ATP recovery and reduces ischemic kidney injury.." Journal of the American Society of Nephrology : JASN, 2011. https://doi.org/10.1681/ASN.2010080808

RethinkPeptides

RethinkPeptides Research Database. "Mitochondria-targeted peptide accelerates ATP recovery and r..." RPEP-01869. Retrieved from https://rethinkpeptides.com/research/szeto-2011-mitochondriatargeted-peptide-accelerates-atp

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Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.