Neuropeptide Autoimmunity at the Blood-Brain Barrier: A New Theory for Neurodegenerative Disease

Autoimmunity against vasoactive neuropeptides (VIP, PACAP) at the blood-brain barrier may contribute to neurodegenerative disease through impaired cerebral blood flow regulation and neuroinflammation.

Staines, D R et al.·Mediators of inflammation·2008·Preliminary EvidenceReview
RPEP-01422ReviewPreliminary Evidence2008RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Review
Evidence
Preliminary Evidence
Sample
Not reported

What This Study Found

Autoimmune targeting of vasoactive neuropeptides (VIP, PACAP) at blood-brain barrier Virchow-Robin spaces may impair cerebral blood flow autoregulation and promote neuroinflammation — a novel immunopathological theory for neurodegenerative diseases.

Key Numbers

How They Did This

review study.

Why This Research Matters

Relevant for neuropeptides, neuroprotection, inflammation.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding Autoimmune targeting of vasoactive neuropeptides (VIP, PACAP) at blood-brain barrier Virchow-Robin spaces may impair cerebral blood flow autoregulatio
Evidence Grade:
preliminary evidence.
Study Age:
Published in 2008.
Original Title:
Postulated role of vasoactive neuropeptide-related immunopathology of the blood brain barrier and Virchow-Robin spaces in the aetiology of neurological-related conditions.
Published In:
Mediators of inflammation, 2008, 792428 (2008)
Database ID:
RPEP-01422

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study

Summarizes existing research on a topic.

What do these levels mean? →

Frequently Asked Questions

What was studied?

Neuropeptide Autoimmunity at the Blood-Brain Barrier: A New Theory for Neurodegenerative Disease

What was found?

Autoimmunity against vasoactive neuropeptides (VIP, PACAP) at the blood-brain barrier may contribute to neurodegenerative disease through impaired cerebral blood flow regulation and neuroinflammation.

Read More on RethinkPeptides

Cite This Study

RPEP-01422·https://rethinkpeptides.com/research/RPEP-01422

APA

Staines, D R; Brenu, E W; Marshall-Gradisnik, S. (2008). Postulated role of vasoactive neuropeptide-related immunopathology of the blood brain barrier and Virchow-Robin spaces in the aetiology of neurological-related conditions.. Mediators of inflammation, 2008, 792428. https://doi.org/10.1155/2008/792428

MLA

Staines, D R, et al. "Postulated role of vasoactive neuropeptide-related immunopathology of the blood brain barrier and Virchow-Robin spaces in the aetiology of neurological-related conditions.." Mediators of inflammation, 2008. https://doi.org/10.1155/2008/792428

RethinkPeptides

RethinkPeptides Research Database. "Postulated role of vasoactive neuropeptide-related immunopat..." RPEP-01422. Retrieved from https://rethinkpeptides.com/research/staines-2008-postulated-role-of-vasoactive

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.