Cerebrolysin reduces excitotoxicity by modulation of cell-death proteins in delayed hours of ischemic reperfusion injury.

Sarode, Lopmudra P et al.·Metabolic brain disease·2023·
RPEP-073492023RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Not classified
Evidence
Not graded
Sample
Not reported

What This Study Found

Key Numbers

How They Did This

Why This Research Matters

What This Study Doesn't Tell Us

Trust & Context

Original Title:
Cerebrolysin reduces excitotoxicity by modulation of cell-death proteins in delayed hours of ischemic reperfusion injury.
Published In:
Metabolic brain disease, 38(7), 2401-2416 (2023)
Database ID:
RPEP-07349

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
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Cite This Study

RPEP-07349·https://rethinkpeptides.com/research/RPEP-07349

APA

Sarode, Lopmudra P; Ghatage, Trupti; Mardhekar, Vishal; Verma, Bhavesh; Prakash, Anand; Ugale, Rajesh R. (2023). Cerebrolysin reduces excitotoxicity by modulation of cell-death proteins in delayed hours of ischemic reperfusion injury.. Metabolic brain disease, 38(7), 2401-2416. https://doi.org/10.1007/s11011-023-01240-4

MLA

Sarode, Lopmudra P, et al. "Cerebrolysin reduces excitotoxicity by modulation of cell-death proteins in delayed hours of ischemic reperfusion injury.." Metabolic brain disease, 2023. https://doi.org/10.1007/s11011-023-01240-4

RethinkPeptides

RethinkPeptides Research Database. "Cerebrolysin reduces excitotoxicity by modulation of cell-de..." RPEP-07349. Retrieved from https://rethinkpeptides.com/research/sarode-2023-cerebrolysin-reduces-excitotoxicity-by

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Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.