Substance P and neurokinin 1 receptor boost the pathogenicity of granulocyte-macrophage colony-stimulating factor-producing T helper cells in dry eye disease.

Rong, Hua et al.·Scandinavian journal of immunology·2025·lowpreclinical
RPEP-13301Preclinicallow2025RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
preclinical
Evidence
low
Sample
N=Not specified (mouse study)
Participants
Mice with induced dry eye disease

What This Study Found

Substance P amplified the pathogenicity of GM-CSF-producing T helper cells in dry eye disease through the NK1R receptor. Blocking NK1R reduced disease severity in a mouse model.

Key Numbers

Substance P increased GM-CSF expression in ThGM cells. NK1R-expressing ThGM transfer significantly exacerbated DED. NK1R-knockdown ThGM weakly aggravated DED. NK2R knockdown had no effect.

How They Did This

Murine dry eye disease model. Characterized NK1R and NK2R expression on ThGM and Th1 cells. Substance P and NKA stimulation assays. Adoptive transfer of NK1R-expressing vs. NK1R-knockdown ThGM cells.

Why This Research Matters

Dry eye disease is an inflammatory condition where neuropeptides and immune cells interact. Identifying the substance P-NK1R axis as a driver opens a new therapeutic target for this common condition.

What This Study Doesn't Tell Us

Mouse model only. DED in humans may involve different immune mechanisms. Adoptive transfer experiments are artificial. NK1R antagonists not tested therapeutically.

Trust & Context

Original Title:
Substance P and neurokinin 1 receptor boost the pathogenicity of granulocyte-macrophage colony-stimulating factor-producing T helper cells in dry eye disease.
Published In:
Scandinavian journal of immunology, 101(1), e13434 (2025)
Database ID:
RPEP-13301

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
What do these levels mean? →

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Cite This Study

RPEP-13301·https://rethinkpeptides.com/research/RPEP-13301

APA

Rong, Hua; Yang, Hai; Liu, Qingqing; Zhang, Hui; Wang, Shaolin. (2025). Substance P and neurokinin 1 receptor boost the pathogenicity of granulocyte-macrophage colony-stimulating factor-producing T helper cells in dry eye disease.. Scandinavian journal of immunology, 101(1), e13434. https://doi.org/10.1111/sji.13434

MLA

Rong, Hua, et al. "Substance P and neurokinin 1 receptor boost the pathogenicity of granulocyte-macrophage colony-stimulating factor-producing T helper cells in dry eye disease.." Scandinavian journal of immunology, 2025. https://doi.org/10.1111/sji.13434

RethinkPeptides

RethinkPeptides Research Database. "Substance P and neurokinin 1 receptor boost the pathogenicit..." RPEP-13301. Retrieved from https://rethinkpeptides.com/research/rong-2025-substance-p-and-neurokinin

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.