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Study breakdown

Both Ghrelin and Des-Acyl Ghrelin Block Fat Cell Fat Breakdown Through a Non-GHS-R Pathway

In VitroPreliminary evidence
The takeaway

Both acyl-ghrelin and des-acyl ghrelin inhibited fat cell lipolysis (fat breakdown) through a receptor different from the classical GHS-R1a, proving des-acyl ghrelin has metabolic activity through its own unknown receptor.

Key finding

Both ghrelin forms inhibited isoproterenol-induced lipolysis in rat adipocytes, with the effect NOT mediated by GHS-R1a (des-acyl ghrelin was equally

What the researchers found

Both ghrelin forms inhibited isoproterenol-induced lipolysis in rat adipocytes, with the effect NOT mediated by GHS-R1a (des-acyl ghrelin was equally effective) — demonstrating fat metabolism regulation through an unidentified non-GHS-R receptor.

Why it matters

Relevant for ghrp, weight-loss, receptor-signaling.

How the study worked

in-vitro study on ghrp, weight-loss.

What this study cannot tell us

See abstract.

How to read the evidence

preliminary evidence.

When this study was published

Published in 2004.

The bigger picture

Advances peptide research.

Questions still open

  • Further research needed.
  • Clinical translation to evaluate.

Common questions

What was studied?
Both Ghrelin and Des-Acyl Ghrelin Block Fat Cell Fat Breakdown Through a Non-GHS-R Pathway
What was found?
Both acyl-ghrelin and des-acyl ghrelin inhibited fat cell lipolysis (fat breakdown) through a receptor different from the classical GHS-R1a, proving des-acyl ghrelin has metabolic activity through its own unknown receptor.

Read the original research

Ghrelin and des-acyl ghrelin both inhibit isoproterenol-induced lipolysis in rat adipocytes via a non-type 1a growth hormone secretagogue receptor.

European journal of pharmacology, 498(1-3), 27-35

Citation

Muccioli, Giampiero; Pons, Nicoletta; Ghè, Corrado; Catapano, Filomena; Granata, Riccarda; Ghigo, Ezio. (2004). Ghrelin and des-acyl ghrelin both inhibit isoproterenol-induced lipolysis in rat adipocytes via a non-type 1a growth hormone secretagogue receptor.. European journal of pharmacology, 498(1-3), 27-35.