Des-acyl ghrelin fragments dilated rat blood vessels by activating potassium channels through a non-GHS-R1a pathway, confirming des-acyl ghrelin has its own vascular effects independent of the classical ghrelin receptor.
Key findingDes-acyl ghrelin fragments dilated rat blood vessels by activating potassium channels through a non-GHS-R1a pathway, confirming des-acyl ghrelin has i
What the researchers found
Des-acyl ghrelin fragments dilated rat blood vessels by activating potassium channels through a non-GHS-R1a pathway, confirming des-acyl ghrelin has its own vascular effects independent of the classical ghrelin receptor.
Why it matters
Relevant for peptide research.
How the study worked
research study.
What this study cannot tell us
See abstract.
How to read the evidence
emerging evidence.
When this study was published
Published in 2009.
The bigger picture
Advances peptide research.
Questions still open
- Further research needed.
Common questions
What was studied?
What was found?
Read the original research
Des-acyl ghrelin fragments evoke endothelium-dependent vasodilatation of rat mesenteric vascular bed via activation of potassium channels.
European journal of pharmacology, 604(1-3), 79-86
Citation
Moazed, Banafsheh; Quest, Dale; Gopalakrishnan, Venkat. (2009). Des-acyl ghrelin fragments evoke endothelium-dependent vasodilatation of rat mesenteric vascular bed via activation of potassium channels.. European journal of pharmacology, 604(1-3), 79-86. https://doi.org/10.1016/j.ejphar.2008.10.032