Spinal Mu-1 Opioid Receptors and Dynorphin B Together Mediate Pain Relief From a Novel Opioid

The antinociceptive effect of synthetic opioid Tyr-d-Arg-Phe-Sar involved both direct mu-1 receptor activation AND endogenous dynorphin B release at the spinal level — a dual-mechanism analgesic.

Mizoguchi, Hirokazu et al.·Peptides·2006·Preliminary EvidenceAnimal StudyAnimal Study
RPEP-01167Animal StudyPreliminary Evidence2006RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Animal Study
Evidence
Preliminary Evidence
Sample
Not reported

What This Study Found

Tyr-d-Arg-Phe-Sar analgesia at the spinal level involved both direct mu-1 opioid receptor activation and endogenous dynorphin B release, with anti-dynorphin antibodies partially blocking the effect — confirming dual direct + endogenous amplification mechanism.

Key Numbers

How They Did This

animal-study study on opioid-peptides, pain.

Why This Research Matters

Relevant for opioid-peptides, pain.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding Tyr-d-Arg-Phe-Sar analgesia at the spinal level involved both direct mu-1 opioid receptor activation and endogenous dynorphin B release, with anti-dyn
Evidence Grade:
preliminary evidence.
Study Age:
Published in 2006.
Original Title:
Contribution of spinal mu(1)-opioid receptors and dynorphin B to the antinociception induced by Tyr-d-Arg-Phe-Sar.
Published In:
Peptides, 27(11), 2786-93 (2006)
Database ID:
RPEP-01167

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / Observational
Case Report / Animal StudyOne case or non-human subjects
This study

Tests effects in animals (usually mice or rats), not humans.

What do these levels mean? →

Frequently Asked Questions

What was studied?

Spinal Mu-1 Opioid Receptors and Dynorphin B Together Mediate Pain Relief From a Novel Opioid

What was found?

The antinociceptive effect of synthetic opioid Tyr-d-Arg-Phe-Sar involved both direct mu-1 receptor activation AND endogenous dynorphin B release at the spinal level — a dual-mechanism analgesic.

Read More on RethinkPeptides

Cite This Study

RPEP-01167·https://rethinkpeptides.com/research/RPEP-01167

APA

Mizoguchi, Hirokazu; Ito, Kanenori; Watanabe, Hiroyuki; Watanabe, Chizuko; Katsuyama, Sou; Fujimura, Tsutomu; Sakurada, Tsukasa; Sakurada, Shinobu. (2006). Contribution of spinal mu(1)-opioid receptors and dynorphin B to the antinociception induced by Tyr-d-Arg-Phe-Sar.. Peptides, 27(11), 2786-93.

MLA

Mizoguchi, Hirokazu, et al. "Contribution of spinal mu(1)-opioid receptors and dynorphin B to the antinociception induced by Tyr-d-Arg-Phe-Sar.." Peptides, 2006.

RethinkPeptides

RethinkPeptides Research Database. "Contribution of spinal mu(1)-opioid receptors and dynorphin ..." RPEP-01167. Retrieved from https://rethinkpeptides.com/research/mizoguchi-2006-contribution-of-spinal-mu1opioid

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.