Mice Without CCK Are Resistant to High-Fat Diet Obesity: The Satiety Hormone's Paradoxical Role

CCK knockout mice were resistant to high-fat diet-induced obesity — paradoxically, losing the satiety hormone CCK protected against weight gain, suggesting CCK has complex metabolic roles beyond simple meal termination.

Lo, Chun-Min et al.·Gastroenterology·2010·
RPEP-016532010RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Not classified
Evidence
Not graded
Sample
Not reported

What This Study Found

CCK knockout mice were resistant to high-fat diet-induced obesity — paradoxically, losing the satiety hormone CCK protected against weight gain, suggesting CCK has complex metabolic roles beyond simple meal termination.

Key Numbers

How They Did This

research study.

Why This Research Matters

Relevant for peptide research.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.

Trust & Context

Key Stat:
Key finding CCK knockout mice were resistant to high-fat diet-induced obesity — paradoxically, losing the satiety hormone CCK protected against weight gain, sugge
Evidence Grade:
emerging evidence.
Study Age:
Published in 2010.
Original Title:
Cholecystokinin knockout mice are resistant to high-fat diet-induced obesity.
Published In:
Gastroenterology, 138(5), 1997-2005 (2010)
Database ID:
RPEP-01653

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
What do these levels mean? →

Frequently Asked Questions

What was studied?

Mice Without CCK Are Resistant to High-Fat Diet Obesity: The Satiety Hormone's Paradoxical Role

What was found?

CCK knockout mice were resistant to high-fat diet-induced obesity — paradoxically, losing the satiety hormone CCK protected against weight gain, suggesting CCK has complex metabolic roles beyond simple meal termination.

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Cite This Study

RPEP-01653·https://rethinkpeptides.com/research/RPEP-01653

APA

Lo, Chun-Min; King, Alexandra; Samuelson, Linda C; Kindel, Tammy Lyn; Rider, Therese; Jandacek, Ronald J; Raybould, Helen E; Woods, Stephen C; Tso, Patrick. (2010). Cholecystokinin knockout mice are resistant to high-fat diet-induced obesity.. Gastroenterology, 138(5), 1997-2005. https://doi.org/10.1053/j.gastro.2010.01.044

MLA

Lo, Chun-Min, et al. "Cholecystokinin knockout mice are resistant to high-fat diet-induced obesity.." Gastroenterology, 2010. https://doi.org/10.1053/j.gastro.2010.01.044

RethinkPeptides

RethinkPeptides Research Database. "Cholecystokinin knockout mice are resistant to high-fat diet..." RPEP-01653. Retrieved from https://rethinkpeptides.com/research/lo-2010-cholecystokinin-knockout-mice-are

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.