BDNF and its TrkB receptor were upregulated in dorsal root ganglia during inflammatory pain in vivo and in vitro, establishing the BDNF-TrkB axis as a contributor to inflammatory pain sensitization.
Key findingBDNF and its TrkB receptor were upregulated in dorsal root ganglia during inflammatory pain in vivo and in vitro, establishing the BDNF-TrkB axis as a
What the researchers found
BDNF and its TrkB receptor were upregulated in dorsal root ganglia during inflammatory pain in vivo and in vitro, establishing the BDNF-TrkB axis as a contributor to inflammatory pain sensitization.
Why it matters
Relevant for peptide research.
How the study worked
research study.
What this study cannot tell us
See abstract.
How to read the evidence
emerging evidence.
When this study was published
Published in 2011.
The bigger picture
Advances peptide research.
Questions still open
- Further research needed.
Common questions
What was studied?
What was found?
Read the original research
Up-regulation of dorsal root ganglia BDNF and trkB receptor in inflammatory pain: an in vivo and in vitro study.
Journal of neuroinflammation, 8, 126
Citation
Lin, Ya-Tin; Ro, Long-Sun; Wang, Hung-Li; Chen, Jin-Chung. (2011). Up-regulation of dorsal root ganglia BDNF and trkB receptor in inflammatory pain: an in vivo and in vitro study.. Journal of neuroinflammation, 8, 126. https://doi.org/10.1186/1742-2094-8-126