Tirzepatide, a dual GIP/GLP-1 receptor agonist, alleviates metabolic dysfunction-associated steatotic liver disease by reducing the expression of CD36 and OBP2A.

Li, Yun et al.·Genes & diseases·2025·
RPEP-121342025RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Not classified
Evidence
Not graded
Sample
Not reported

What This Study Found

Key Numbers

How They Did This

Why This Research Matters

What This Study Doesn't Tell Us

Trust & Context

Original Title:
Tirzepatide, a dual GIP/GLP-1 receptor agonist, alleviates metabolic dysfunction-associated steatotic liver disease by reducing the expression of CD36 and OBP2A.
Published In:
Genes & diseases, 12(6), 101761 (2025)
Database ID:
RPEP-12134

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
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Cite This Study

RPEP-12134·https://rethinkpeptides.com/research/RPEP-12134

APA

Li, Yun; Sun, Wencong; Liu, Hong; Ruan, Xiong Z. (2025). Tirzepatide, a dual GIP/GLP-1 receptor agonist, alleviates metabolic dysfunction-associated steatotic liver disease by reducing the expression of CD36 and OBP2A.. Genes & diseases, 12(6), 101761. https://doi.org/10.1016/j.gendis.2025.101761

MLA

Li, Yun, et al. "Tirzepatide, a dual GIP/GLP-1 receptor agonist, alleviates metabolic dysfunction-associated steatotic liver disease by reducing the expression of CD36 and OBP2A.." Genes & diseases, 2025. https://doi.org/10.1016/j.gendis.2025.101761

RethinkPeptides

RethinkPeptides Research Database. "Tirzepatide, a dual GIP/GLP-1 receptor agonist, alleviates m..." RPEP-12134. Retrieved from https://rethinkpeptides.com/research/li-2025-tirzepatide-a-dual-gipglp1

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Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.