Ghrelin Protects Blood Vessel Cells by Blocking Inflammation Through NF-κB Inhibition

Ghrelin directly inhibited TNF-α-induced pro-inflammatory responses and NF-κB nuclear translocation in human endothelial cells, revealing a specific molecular mechanism for ghrelin's cardiovascular protective anti-inflammatory effect.

Li, Wei Gen et al.·Circulation·2004·Moderate Evidencein-vitro
RPEP-00940In VitroModerate Evidence2004RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
in-vitro
Evidence
Moderate Evidence
Sample
Not reported

What This Study Found

Ghrelin inhibited TNF-α-induced VCAM-1, ICAM-1, and MCP-1 expression in human endothelial cells by blocking NF-κB nuclear translocation via IκBα preservation — a specific anti-inflammatory mechanism protecting blood vessels from atherosclerosis.

Key Numbers

How They Did This

in-vitro study examining ghrp and cardiovascular.

Why This Research Matters

Advances understanding of ghrp, cardiovascular, inflammation, receptor-signaling.

The Bigger Picture

Contributes to peptide research with clinical implications.

What This Study Doesn't Tell Us

Study-specific limitations; see abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation potential to evaluate.

Trust & Context

Key Stat:
Key finding Ghrelin inhibited TNF-α-induced VCAM-1, ICAM-1, and MCP-1 expression in human endothelial cells by blocking NF-κB nuclear translocation via IκBα prese
Evidence Grade:
moderate evidence from in-vitro study.
Study Age:
Published in 2004.
Original Title:
Ghrelin inhibits proinflammatory responses and nuclear factor-kappaB activation in human endothelial cells.
Published In:
Circulation, 109(18), 2221-6 (2004)
Database ID:
RPEP-00940

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / ObservationalSnapshot without intervening
This study
Case Report / Animal Study
What do these levels mean? →

Frequently Asked Questions

What was studied?

Ghrelin Protects Blood Vessel Cells by Blocking Inflammation Through NF-κB Inhibition

What was found?

Ghrelin directly inhibited TNF-α-induced pro-inflammatory responses and NF-κB nuclear translocation in human endothelial cells, revealing a specific molecular mechanism for ghrelin's cardiovascular protective anti-inflammatory effect.

Read More on RethinkPeptides

Cite This Study

RPEP-00940·https://rethinkpeptides.com/research/RPEP-00940

APA

Li, Wei Gen; Gavrila, Dan; Liu, Xuebo; Wang, Lixing; Gunnlaugsson, Skuli; Stoll, Lynn L; McCormick, Michael L; Sigmund, Curt D; Tang, Chaosu; Weintraub, Neal L. (2004). Ghrelin inhibits proinflammatory responses and nuclear factor-kappaB activation in human endothelial cells.. Circulation, 109(18), 2221-6.

MLA

Li, Wei Gen, et al. "Ghrelin inhibits proinflammatory responses and nuclear factor-kappaB activation in human endothelial cells.." Circulation, 2004.

RethinkPeptides

RethinkPeptides Research Database. "Ghrelin inhibits proinflammatory responses and nuclear facto..." RPEP-00940. Retrieved from https://rethinkpeptides.com/research/li-2004-ghrelin-inhibits-proinflammatory-responses

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.