GI hormones (GLP-1, GIP, CCK, gastrin) regulate pancreatic beta-cell mass through proliferation and anti-apoptosis, explaining how incretin drugs may preserve insulin production capacity in diabetes.
Key findingGI hormones (GLP-1, GIP, CCK, gastrin) regulate pancreatic beta-cell mass through proliferation and anti-apoptosis, explaining how incretin drugs may
What the researchers found
GI hormones (GLP-1, GIP, CCK, gastrin) regulate pancreatic beta-cell mass through proliferation and anti-apoptosis, explaining how incretin drugs may preserve insulin production capacity in diabetes.
Why it matters
Relevant for peptide research.
How the study worked
research study.
What this study cannot tell us
See abstract.
How to read the evidence
emerging evidence.
When this study was published
Published in 2010.
The bigger picture
Advances peptide research.
Questions still open
- Further research needed.
Common questions
What was studied?
What was found?
Read the original research
Gastrointestinal hormones and the regulation of β-cell mass.
Annals of the New York Academy of Sciences, 1212, 41-58
Citation
Lavine, Jeremy A; Attie, Alan D. (2010). Gastrointestinal hormones and the regulation of β-cell mass.. Annals of the New York Academy of Sciences, 1212, 41-58. https://doi.org/10.1111/j.1749-6632.2010.05802.x