A small peptide antagonist of the Fas receptor inhibits neuroinflammation and prevents axon degeneration and retinal ganglion cell death in an inducible mouse model of glaucoma.
Quick Facts
What This Study Found
Key Numbers
How They Did This
Why This Research Matters
What This Study Doesn't Tell Us
Trust & Context
- Original Title:
- A small peptide antagonist of the Fas receptor inhibits neuroinflammation and prevents axon degeneration and retinal ganglion cell death in an inducible mouse model of glaucoma.
- Published In:
- Journal of neuroinflammation, 16(1), 184 (2019)
- Authors:
- Krishnan, Anitha, Kocab, Andrew J, Zacks, David N, Marshak-Rothstein, Ann, Gregory-Ksander, Meredith
- Database ID:
- RPEP-04292
Evidence Hierarchy
Read More on RethinkPeptides
Related articles coming soon.
Cite This Study
https://rethinkpeptides.com/research/RPEP-04292APA
Krishnan, Anitha; Kocab, Andrew J; Zacks, David N; Marshak-Rothstein, Ann; Gregory-Ksander, Meredith. (2019). A small peptide antagonist of the Fas receptor inhibits neuroinflammation and prevents axon degeneration and retinal ganglion cell death in an inducible mouse model of glaucoma.. Journal of neuroinflammation, 16(1), 184. https://doi.org/10.1186/s12974-019-1576-3
MLA
Krishnan, Anitha, et al. "A small peptide antagonist of the Fas receptor inhibits neuroinflammation and prevents axon degeneration and retinal ganglion cell death in an inducible mouse model of glaucoma.." Journal of neuroinflammation, 2019. https://doi.org/10.1186/s12974-019-1576-3
RethinkPeptides
RethinkPeptides Research Database. "A small peptide antagonist of the Fas receptor inhibits neur..." RPEP-04292. Retrieved from https://rethinkpeptides.com/research/krishnan-2019-a-small-peptide-antagonist
Access the Original Study
Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.
This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.