Chronic Social Stress Changes Brain CRF Levels: The Neural Basis of Stress-Induced Anxiety

Chronic psychosocial stress altered CRF levels in the paraventricular nucleus and central amygdala of rats, with changes correlating with anxiety-like behavior — mapping the neural basis of stress-induced anxiety disorders.

Kozicz, T et al.·Psychoneuroendocrinology·2008·Preliminary EvidenceAnimal StudyAnimal Study
RPEP-01368Animal StudyPreliminary Evidence2008RETHINKTHC RESEARCH DATABASErethinkthc.com/research

Quick Facts

Study Type
Animal Study
Evidence
Preliminary Evidence
Sample
Not reported

What This Study Found

Chronic psychosocial stress produced CRF alterations in the PVN and central amygdala correlating with increased anxiety-like behavior, mapping how chronic social stress reprograms the brain's CRF stress system to produce persistent anxiety.

Key Numbers

How They Did This

animal-study study.

Why This Research Matters

Relevant for neuropeptides, anxiety-mood.

The Bigger Picture

Advances peptide research.

What This Study Doesn't Tell Us

See abstract.

Questions This Raises

  • ?Further research needed.
  • ?Clinical translation to evaluate.

Trust & Context

Key Stat:
Key finding Chronic psychosocial stress produced CRF alterations in the PVN and central amygdala correlating with increased anxiety-like behavior, mapping how chr
Evidence Grade:
preliminary evidence.
Study Age:
Published in 2008.
Original Title:
Chronic psychosocial stress affects corticotropin-releasing factor in the paraventricular nucleus and central extended amygdala as well as urocortin 1 in the non-preganglionic Edinger-Westphal nucleus of the tree shrew.
Published In:
Psychoneuroendocrinology, 33(6), 741-54 (2008)
Database ID:
RPEP-01368

Evidence Hierarchy

Meta-Analysis / Systematic Review
Randomized Controlled Trial
Cohort / Case-Control
Cross-Sectional / Observational
Case Report / Animal StudyOne case or non-human subjects
This study

Tests effects in animals (usually mice or rats), not humans.

What do these levels mean? →

Frequently Asked Questions

What was studied?

Chronic Social Stress Changes Brain CRF Levels: The Neural Basis of Stress-Induced Anxiety

What was found?

Chronic psychosocial stress altered CRF levels in the paraventricular nucleus and central amygdala of rats, with changes correlating with anxiety-like behavior — mapping the neural basis of stress-induced anxiety disorders.

Read More on RethinkPeptides

Cite This Study

RPEP-01368·https://rethinkpeptides.com/research/RPEP-01368

APA

Kozicz, T; Bordewin, L A P; Czéh, B; Fuchs, E; Roubos, E W. (2008). Chronic psychosocial stress affects corticotropin-releasing factor in the paraventricular nucleus and central extended amygdala as well as urocortin 1 in the non-preganglionic Edinger-Westphal nucleus of the tree shrew.. Psychoneuroendocrinology, 33(6), 741-54. https://doi.org/10.1016/j.psyneuen.2008.02.012

MLA

Kozicz, T, et al. "Chronic psychosocial stress affects corticotropin-releasing factor in the paraventricular nucleus and central extended amygdala as well as urocortin 1 in the non-preganglionic Edinger-Westphal nucleus of the tree shrew.." Psychoneuroendocrinology, 2008. https://doi.org/10.1016/j.psyneuen.2008.02.012

RethinkPeptides

RethinkPeptides Research Database. "Chronic psychosocial stress affects corticotropin-releasing ..." RPEP-01368. Retrieved from https://rethinkpeptides.com/research/kozicz-2008-chronic-psychosocial-stress-affects

Access the Original Study

Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.

This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.