Anti-nerve growth factor immunoglobulin-induced pain relief was partly mediated through enhanced endogenous opioid peptide signaling, linking NGF antagonism to the opioid pain control system.
Key findingAnti-nerve growth factor immunoglobulin-induced pain relief was partly mediated through enhanced endogenous opioid peptide signaling, linking NGF anta
What the researchers found
Anti-nerve growth factor immunoglobulin-induced pain relief was partly mediated through enhanced endogenous opioid peptide signaling, linking NGF antagonism to the opioid pain control system.
Why it matters
Relevant for peptide research.
How the study worked
research study.
What this study cannot tell us
See abstract.
How to read the evidence
emerging evidence.
When this study was published
Published in 2011.
The bigger picture
Advances peptide research.
Questions still open
- Further research needed.
Common questions
What was studied?
What was found?
Read the original research
Potential mechanisms for hypoalgesia induced by anti-nerve growth factor immunoglobulin are identified using autoimmune nerve growth factor deprivation.
Neuroscience, 193, 452-65
Citation
Hoffman, E M; Zhang, Z; Anderson, M B; Schechter, R; Miller, K E. (2011). Potential mechanisms for hypoalgesia induced by anti-nerve growth factor immunoglobulin are identified using autoimmune nerve growth factor deprivation.. Neuroscience, 193, 452-65. https://doi.org/10.1016/j.neuroscience.2011.06.069