Anti-NGF Antibody Pain Relief Involves Changes in Endogenous Opioid Peptide Systems
Anti-nerve growth factor immunoglobulin-induced pain relief was partly mediated through enhanced endogenous opioid peptide signaling, linking NGF antagonism to the opioid pain control system.
Quick Facts
What This Study Found
Anti-nerve growth factor immunoglobulin-induced pain relief was partly mediated through enhanced endogenous opioid peptide signaling, linking NGF antagonism to the opioid pain control system.
Key Numbers
How They Did This
research study.
Why This Research Matters
Relevant for peptide research.
The Bigger Picture
Advances peptide research.
What This Study Doesn't Tell Us
See abstract.
Questions This Raises
- ?Further research needed.
Trust & Context
- Key Stat:
- Key finding Anti-nerve growth factor immunoglobulin-induced pain relief was partly mediated through enhanced endogenous opioid peptide signaling, linking NGF anta
- Evidence Grade:
- emerging evidence.
- Study Age:
- Published in 2011.
- Original Title:
- Potential mechanisms for hypoalgesia induced by anti-nerve growth factor immunoglobulin are identified using autoimmune nerve growth factor deprivation.
- Published In:
- Neuroscience, 193, 452-65 (2011)
- Authors:
- Hoffman, E M, Zhang, Z, Anderson, M B, Schechter, R, Miller, K E
- Database ID:
- RPEP-01780
Evidence Hierarchy
Frequently Asked Questions
What was studied?
Anti-NGF Antibody Pain Relief Involves Changes in Endogenous Opioid Peptide Systems
What was found?
Anti-nerve growth factor immunoglobulin-induced pain relief was partly mediated through enhanced endogenous opioid peptide signaling, linking NGF antagonism to the opioid pain control system.
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Cite This Study
https://rethinkpeptides.com/research/RPEP-01780APA
Hoffman, E M; Zhang, Z; Anderson, M B; Schechter, R; Miller, K E. (2011). Potential mechanisms for hypoalgesia induced by anti-nerve growth factor immunoglobulin are identified using autoimmune nerve growth factor deprivation.. Neuroscience, 193, 452-65. https://doi.org/10.1016/j.neuroscience.2011.06.069
MLA
Hoffman, E M, et al. "Potential mechanisms for hypoalgesia induced by anti-nerve growth factor immunoglobulin are identified using autoimmune nerve growth factor deprivation.." Neuroscience, 2011. https://doi.org/10.1016/j.neuroscience.2011.06.069
RethinkPeptides
RethinkPeptides Research Database. "Potential mechanisms for hypoalgesia induced by anti-nerve g..." RPEP-01780. Retrieved from https://rethinkpeptides.com/research/hoffman-2011-potential-mechanisms-for-hypoalgesia
Access the Original Study
Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.
This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.