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Study breakdown

All Three GLP-1 Drugs Work in MC4R Knockout Mice — Even Without the Melanocortin Pathway

evidence
The takeaway

Semaglutide (19.7%), tirzepatide (31.6%), and retatrutide (24.1%) all produced significant weight loss in MC4R KO mice, proving they work independently of the melanocortin obesity pathway.

31.6% weight loss without MC4R

Tirzepatide achieved the largest weight loss even in mice completely lacking the melanocortin obesity pathway

What the researchers found

Weight loss in MC4R KO mice: semaglutide 19.7%, tirzepatide 31.6%, retatrutide 24.1%. All improved insulin, HOMA-IR, cholesterol, AST/ALT, and suppressed FA synthesis genes. Only tirzepatide reduced RQ. No effect on inflammation genes.

Why it matters

MC4R deficiency causes 5% of severe human obesity. Proving GLP-1 drugs work even without this pathway provides hope for genetically obese patients.

How the study worked

21-day treatment of MC4R KO mice with semaglutide, tirzepatide, or retatrutide, with body composition (Echo-MRI), metabolic parameters, HOMA-IR, liver enzyme/gene expression analysis, and indirect calorimetry.

What this study cannot tell us

Mouse model. 21-day treatment is short. MC4R KO mice don't perfectly replicate human MC4R heterozygous deficiency. All drugs reduced lean mass (a concern).

How to read the evidence

Well-controlled preclinical study with three drug comparisons. Validates MC4R KO mice as a model and GLP-1 drug mechanism independence.

When this study was published

Published in 2025.

The bigger picture

GLP-1 drugs work through mechanisms independent of the melanocortin pathway, suggesting they could be effective first-line therapy even for genetic forms of obesity.

Questions still open

  • Would these drugs show similar efficacy in humans with MC4R mutations?
  • Does tirzepatide's superior efficacy come from GIP co-agonism in the MC4R-deficient context?
  • Should genetic obesity patients be prioritized for GLP-1 therapy?

Common questions

Do GLP-1 drugs work for genetic obesity?
Yes. This study proved semaglutide, tirzepatide, and retatrutide all cause significant weight loss even in mice that completely lack the MC4R melanocortin pathway — the most common cause of genetic obesity.
Which drug worked best in genetic obesity mice?
Tirzepatide (31.6% weight loss) outperformed retatrutide (24.1%) and semaglutide (19.7%), suggesting the GIP receptor co-activation provides extra benefit when the melanocortin pathway is absent.

Read the original research

Efficacy of GLP-1 analog peptides, semaglutide, tirzepatide, and retatrutide on MC4R deficient obesity and their comparison.

International journal of obesity (2005)

Citation

Hitaka, Kosuke; Sugawara, Takumi; Matsumoto, Mitsuharu; Nio, Yasunori. (2026). Efficacy of GLP-1 analog peptides, semaglutide, tirzepatide, and retatrutide on MC4R deficient obesity and their comparison.. International journal of obesity (2005). https://doi.org/10.1038/s41366-026-02025-2