VIP and VPAC receptor expression in salivary glands suggests VIP's neuroprotective/anti-inflammatory role contributes to gland function, with VIP dysregulation potentially involved in Sjögren's syndrome pathogenesis.
Key findingVIP and VPAC receptor expression in salivary glands suggests VIP's neuroprotective/anti-inflammatory role contributes to gland function, with VIP dysr
What the researchers found
VIP and VPAC receptor expression in salivary glands suggests VIP's neuroprotective/anti-inflammatory role contributes to gland function, with VIP dysregulation potentially involved in Sjögren's syndrome pathogenesis.
Why it matters
Relevant for peptide research.
How the study worked
research study.
What this study cannot tell us
See abstract.
How to read the evidence
emerging evidence.
When this study was published
Published in 2011.
The bigger picture
Advances peptide research.
Questions still open
- Further research needed.
Common questions
What was studied?
What was found?
Read the original research
Vasoactive intestinal peptide/vasoactive intestinal peptide receptor relative expression in salivary glands as one endogenous modulator of acinar cell apoptosis in a murine model of Sjögren's syndrome.
Clinical and experimental immunology, 166(3), 309-16
Citation
Hauk, V; Calafat, M; Larocca, L; Fraccaroli, L; Grasso, E; Ramhorst, R; Leirós, C Pérez. (2011). Vasoactive intestinal peptide/vasoactive intestinal peptide receptor relative expression in salivary glands as one endogenous modulator of acinar cell apoptosis in a murine model of Sjögren's syndrome.. Clinical and experimental immunology, 166(3), 309-16. https://doi.org/10.1111/j.1365-2249.2011.04478.x