Cerebrolysin protects PC12 cells from CoCl2-induced hypoxia employing GSK3β signaling.
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What This Study Found
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How They Did This
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What This Study Doesn't Tell Us
Trust & Context
- Original Title:
- Cerebrolysin protects PC12 cells from CoCl2-induced hypoxia employing GSK3β signaling.
- Published In:
- International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience, 38, 52-8 (2014)
- Authors:
- Hartwig, Kerstin, Fackler, Viktoria, Jaksch-Bogensperger, Heidi, Winter, Stefan, Furtner, Tanja, Couillard-Despres, Sebastien, Meier, Dieter, Moessler, Herbert, Aigner, Ludwig
- Database ID:
- RPEP-02400
Evidence Hierarchy
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Cite This Study
https://rethinkpeptides.com/research/RPEP-02400APA
Hartwig, Kerstin; Fackler, Viktoria; Jaksch-Bogensperger, Heidi; Winter, Stefan; Furtner, Tanja; Couillard-Despres, Sebastien; Meier, Dieter; Moessler, Herbert; Aigner, Ludwig. (2014). Cerebrolysin protects PC12 cells from CoCl2-induced hypoxia employing GSK3β signaling.. International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience, 38, 52-8. https://doi.org/10.1016/j.ijdevneu.2014.07.005
MLA
Hartwig, Kerstin, et al. "Cerebrolysin protects PC12 cells from CoCl2-induced hypoxia employing GSK3β signaling.." International journal of developmental neuroscience : the official journal of the International Society for Developmental Neuroscience, 2014. https://doi.org/10.1016/j.ijdevneu.2014.07.005
RethinkPeptides
RethinkPeptides Research Database. "Cerebrolysin protects PC12 cells from CoCl2-induced hypoxia ..." RPEP-02400. Retrieved from https://rethinkpeptides.com/research/hartwig-2014-cerebrolysin-protects-pc12-cells
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Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.
This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.