GHRH and its synthetic agonists accelerated wound healing through direct cellular effects (fibroblast proliferation, collagen synthesis) and GH/IGF-1 mediation — a new wound healing application for the GH-axis.
Key findingGHRH and its synthetic agonists accelerated wound healing through direct cellular effects (fibroblast proliferation, collagen synthesis) and GH/IGF-1
What the researchers found
GHRH and its synthetic agonists accelerated wound healing through direct cellular effects (fibroblast proliferation, collagen synthesis) and GH/IGF-1 mediation — a new wound healing application for the GH-axis.
Why it matters
Relevant for peptide research.
How the study worked
research study.
What this study cannot tell us
See abstract.
How to read the evidence
emerging evidence.
When this study was published
Published in 2010.
The bigger picture
Advances peptide research.
Questions still open
- Further research needed.
Common questions
What was studied?
What was found?
Read the original research
Acceleration of wound healing by growth hormone-releasing hormone and its agonists.
Proceedings of the National Academy of Sciences of the United States of America, 107(43), 18611-5
Citation
Dioufa, Nikolina; Schally, Andrew V; Chatzistamou, Ioulia; Moustou, Evi; Block, Norman L; Owens, Gary K; Papavassiliou, Athanasios G; Kiaris, Hippokratis. (2010). Acceleration of wound healing by growth hormone-releasing hormone and its agonists.. Proceedings of the National Academy of Sciences of the United States of America, 107(43), 18611-5. https://doi.org/10.1073/pnas.1013942107