Inflammatory signals directly regulate cardiac natriuretic peptide gene expression and secretion, explaining why BNP rises in inflammatory conditions beyond heart failure — inflammation-cardiac peptide cross-talk.
Key findingInflammatory signals directly regulate cardiac natriuretic peptide gene expression and secretion, explaining why BNP rises in inflammatory conditions
What the researchers found
Inflammatory signals directly regulate cardiac natriuretic peptide gene expression and secretion, explaining why BNP rises in inflammatory conditions beyond heart failure — inflammation-cardiac peptide cross-talk.
Why it matters
Relevant for peptide research.
How the study worked
research study.
What this study cannot tell us
See abstract.
How to read the evidence
emerging evidence.
When this study was published
Published in 2009.
The bigger picture
Advances peptide research.
Questions still open
- Further research needed.
Common questions
What was studied?
What was found?
Read the original research
Cardiac natriuretic peptides gene expression and secretion in inflammation.
Journal of investigative medicine : the official publication of the American Federation for Clinical Research, 57(1), 29-32
Citation
de Bold, Adolfo J. (2009). Cardiac natriuretic peptides gene expression and secretion in inflammation.. Journal of investigative medicine : the official publication of the American Federation for Clinical Research, 57(1), 29-32. https://doi.org/10.2310/JIM.0b013e3181948b37