The human cathelicidin LL-37 preferentially promotes apoptosis of infected airway epithelium.
Quick Facts
What This Study Found
Key Numbers
How They Did This
Why This Research Matters
What This Study Doesn't Tell Us
Trust & Context
- Original Title:
- The human cathelicidin LL-37 preferentially promotes apoptosis of infected airway epithelium.
- Published In:
- American journal of respiratory cell and molecular biology, 43(6), 692-702 (2010)
- Authors:
- Barlow, Peter G, Beaumont, Paula E, Cosseau, Celine, Mackellar, Annie, Wilkinson, Thomas S, Hancock, Robert E W, Haslett, Chris, Govan, John R W, Simpson, A John, Davidson, Donald J
- Database ID:
- RPEP-01585
Evidence Hierarchy
Read More on RethinkPeptides
Related articles coming soon.
Cite This Study
https://rethinkpeptides.com/research/RPEP-01585APA
Barlow, Peter G; Beaumont, Paula E; Cosseau, Celine; Mackellar, Annie; Wilkinson, Thomas S; Hancock, Robert E W; Haslett, Chris; Govan, John R W; Simpson, A John; Davidson, Donald J. (2010). The human cathelicidin LL-37 preferentially promotes apoptosis of infected airway epithelium.. American journal of respiratory cell and molecular biology, 43(6), 692-702. https://doi.org/10.1165/rcmb.2009-0250OC
MLA
Barlow, Peter G, et al. "The human cathelicidin LL-37 preferentially promotes apoptosis of infected airway epithelium.." American journal of respiratory cell and molecular biology, 2010. https://doi.org/10.1165/rcmb.2009-0250OC
RethinkPeptides
RethinkPeptides Research Database. "The human cathelicidin LL-37 preferentially promotes apoptos..." RPEP-01585. Retrieved from https://rethinkpeptides.com/research/barlow-2010-the-human-cathelicidin-ll37
Access the Original Study
Study data sourced from PubMed, a service of the U.S. National Library of Medicine, National Institutes of Health.
This study breakdown was produced by the RethinkPeptides research team. We analyze and report published research findings without making health recommendations. All interpretations are based solely on the published abstract and study data.