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Study breakdown

Oxyntomodulin, GLP-1, and Glucagon All Cut Food Intake Equally — But Combining Them Didn't Help More

Clinical TrialModerate evidence
The takeaway

Oxyntomodulin, GLP-1, and glucagon each reduced food intake by about 15% in healthy men, but combining GLP-1 and glucagon provided no additional benefit, challenging assumptions about dual-receptor weight loss strategies.

No additive effect

GLP-1+glucagon combined reduced food intake by the same amount (~15%) as either peptide alone in 15 healthy men

What the researchers found

In a rigorous head-to-head comparison, oxyntomodulin, GLP-1, glucagon, and GLP-1+glucagon combined all reduced food intake by similar amounts compared to saline (roughly 15-17% reduction). However, combining GLP-1 and glucagon together did not produce an additive effect — the combination was no better than either peptide alone.

The mechanisms differed: oxyntomodulin, GLP-1, and GLP-1+glucagon slowed gastric emptying and reduced appetite scores, while glucagon reduced food intake without affecting either gastric emptying or appetite. No peptide infusion significantly changed resting energy expenditure compared to saline.

Why it matters

Oxyntomodulin activates both GLP-1 and glucagon receptors, and the drug industry has bet heavily on dual-agonists (like survodutide) that mimic this. This study challenges a key assumption: that activating both receptors simultaneously produces additive benefits. The finding that GLP-1+glucagon was no better than either alone at reducing food intake suggests oxyntomodulin's weight-loss mechanism may not be simply the sum of its receptor activities — there may be something else at play.

The numbers in context

n=15 healthy males · Age 22 (18-32) · BMI 23 (21-26) · GLP-1: 1 pmol/kg/min · Glucagon: 0.86 pmol/kg/min · Oxyntomodulin: 3 pmol/kg/min · Food intake (g): saline 811, GLP-1 669, glucagon 686, OXM 689, GLP-1+glucagon 688 · No REE changes

How the study worked

Double-blinded, randomized, crossover study in 15 healthy young men. Each participant received five 4-hour liquid meal tests during infusion of saline, GLP-1, glucagon, oxyntomodulin, or GLP-1+glucagon on separate occasions. Researchers measured resting energy expenditure (oxygen uptake), gastric emptying, composite appetite scores, and ad libitum food intake.

Who was studied

15 young healthy male volunteers, aged 18-32 years, BMI 21-26 kg/m²

What this study cannot tell us

Small sample size (n=15) of only young, lean, healthy men — results may differ in obese individuals or women. Acute infusion study (4 hours) may not reflect chronic effects relevant to weight management. The doses tested may not have been optimal to demonstrate additive effects. Only one dose combination of GLP-1+glucagon was tested.

How to read the evidence

This is a moderate-quality human study using a rigorous double-blind, randomized, crossover design — the gold standard for within-subject comparisons. Published in the Journal of Clinical Endocrinology & Metabolism (a top journal). However, the small sample (n=15) of healthy lean men and acute dosing protocol limit generalizability.

When this study was published

Published in 2015, this study predates the current wave of dual-agonist drugs in development. Its findings remain highly relevant as GLP-1/glucagon dual agonists advance through clinical trials.

The bigger picture

The pharmaceutical industry has invested billions in dual GLP-1/glucagon agonists (like survodutide, efinopegdutide, and mazdutide) based on the theory that hitting both receptors simultaneously produces superior metabolic effects. This study's finding that combining GLP-1 and glucagon infusions didn't produce additive appetite suppression complicates that narrative. It suggests the clinical benefits of dual agonists may come from mechanisms beyond simple receptor co-activation — perhaps from receptor interactions, timing, or downstream signaling differences.

Questions still open

  • Would combining GLP-1 and glucagon show additive effects on food intake at different doses, in obese subjects, or over longer treatment durations?
  • If oxyntomodulin's weight-loss effect isn't simply dual receptor activation, what additional mechanisms does it employ?
  • Does glucagon's ability to reduce food intake without affecting appetite or gastric emptying suggest a distinct central mechanism?

Common questions

What is oxyntomodulin and why is it important for weight loss?
Oxyntomodulin is a peptide hormone naturally produced in your gut after eating. It activates both the GLP-1 receptor (which suppresses appetite) and the glucagon receptor (which increases energy burning). Several drug companies are developing synthetic versions and dual-agonist drugs inspired by oxyntomodulin for weight loss.
Why is it surprising that combining GLP-1 and glucagon wasn't more effective?
The entire rationale for dual GLP-1/glucagon agonist drugs — now in late-stage clinical trials — is based on the assumption that activating both receptors is better than one. This study showed that, at least for acute food intake reduction, combining the two peptides was no better than either alone, suggesting the dual-agonist benefit may come from something other than simple additive effects.

Read the original research

Effect of Oxyntomodulin, Glucagon, GLP-1, and Combined Glucagon +GLP-1 Infusion on Food Intake, Appetite, and Resting Energy Expenditure.

The Journal of clinical endocrinology and metabolism, 100(12), 4541-52

Citation

Bagger, Jonatan Ising; Holst, Jens Juul; Hartmann, Bolette; Andersen, Birgitte; Knop, Filip Krag; Vilsbøll, Tina. (2015). Effect of Oxyntomodulin, Glucagon, GLP-1, and Combined Glucagon +GLP-1 Infusion on Food Intake, Appetite, and Resting Energy Expenditure.. The Journal of clinical endocrinology and metabolism, 100(12), 4541-52. https://doi.org/10.1210/jc.2015-2335